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Author ORCID Identifier

https://orcid.org/0000-0002-8887-4487

Date Available

3-25-2028

Year of Publication

2026

Document Type

Doctoral Dissertation

Degree Name

Doctor of Philosophy (PhD)

College

Health Sciences

Department/School/Program

Rehabilitation Sciences

Faculty

Esther Dupont-Versteegden

Faculty

Timothy Butterfield

Abstract

Skeletal muscle adapts to mechanical load by hypertrophy with use and atrophy with disuse. However, sex-specific differences in these responses, particularly during recovery, are poorly understood. This dissertation investigated how male and female rats respond to altered mechanical loading using hindlimb suspension (HS) to model disuse, reambulation (RA) for recovery, and mechanotherapy as an intervention. In study 1, females exhibited less atrophy during HS than males, associated with a smaller decline in myofibrillar protein synthesis (Ksyn). During recovery, females showed increased Ksyn, whereas males demonstrated reduced protein degradation (Kdeg). Mechanotherapy did not significantly alter fiber type, satellite cell, or myonuclear number. Study 2 used RNA sequencing to assess transcriptional responses. During HS, females showed greater metabolic gene expression, while males displayed increased inflammatory signaling and reduced mitochondrial gene expression. With recovery, females largely reversed disuse-induced changes and increased RNA synthesis. In contrast, males exhibited greater extracellular matrix remodeling, reduced ribosomal biogenesis, and decreased RNA degradation. Study 3 examined early (8-hour) recovery and single-cell transcription. Females showed more early gene changes, while fibro-adipogenic progenitors demonstrated stronger extracellular remodeling signatures in males. Overall, males and females differ in molecular responses to disuse and recovery, emphasizing the need for sex-specific therapeutic targets.

Digital Object Identifier (DOI)

https://doi.org/10.13023/etd.2026.20

Archival?

Archival

Funding Information

These studies were supported by the National Institutes of Health grants AT009268, AT009268-02S1, AT009268-05S1

Available for download on Saturday, March 25, 2028

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