Authors

Lijun Ma, Wake Forest School of Medicine
Mariana Murea, Wake Forest School of Medicine
James A. Snipes, Wake Forest School of Medicine
Alejandra Marinelarena, National Institute of Diabetes and Digestive and Kidney Diseases
Jacqueline Krüger, University of Leipzig, German
Pamela J. Hicks, Wake Forest School of Medicine
Kurt A. Langberg, Wake Forest School of Medicine
Meredith A. Bostrom, Wake Forest School of Medicine
Jessica N. Cooke, Wake Forest School of Medicine
Daisuke Suzuki, Tokai University, Japan
Tetsuya Babazono, Tokyo Women's Medical University, Japan
Takashi Uzu, Shiga University of Medical Science, Japan
Sydney C. W. Tang, University of Hong Kong, China
Ashis K. Mondal, Wake Forest School of Medicine
Neeraj K. Sharma, Wake Forest School of Medicine
Sayuko Kobes, National Institute of Diabetes and Digestive and Kidney Diseases
Peter A. Antinozzi, Wake Forest School of Medicine
Matthew Davis, Wake Forest School of Medicine
Swapan K. Das, Wake Forest School of Medicine
Neda Rasouli, University of Colorado
Philip A. Kern, University of KentuckyFollow
Nathan J. Shores, Wake Forest School of Medicine
Lawrence L. Rudel, Wake Forest School of Medicine
Matthias Blüher, University of Leipzig, German
Michael Stumvoll, University of Leipzig, German
Donald W. Bowden, Wake Forest School of Medicine
Shiro Maeda, RIKEN Center for Genomic Medicine, Japan
John S. Parks, Wake Forest School of Medicine
Peter Kovacs, University of Leipzig, German
Robert L. Hanson, National Institute of Diabetes and Digestive and Kidney Diseases
Leslie J. Baier, National Institute of Diabetes and Digestive and Kidney Diseases
Steven C. Elbein, Wake Forest School of Medicine
Barry I. Freedman, Wake Forest School of Medicine

Abstract

Acetyl coenzyme A carboxylase B gene (ACACB) single nucleotide polymorphism (SNP) rs2268388 is reproducibly associated with type 2 diabetes (T2DM)-associated nephropathy (DN). ACACB knock-out mice are also protected from obesity. This study assessed relationships between rs2268388, body mass index (BMI) and gene expression in multiple populations, with and without T2DM. Among subjects without T2DM, rs2268388 DN risk allele (T) associated with higher BMI in Pima Indian children (n = 2021; p-additive = 0.029) and African Americans (AAs) (n = 177; p-additive = 0.05), with a trend in European Americans (EAs) (n = 512; p-additive = 0.09), but not Germans (n = 858; p-additive = 0.765). Association with BMI was seen in a meta-analysis including all non-T2DM subjects (n = 3568; p-additive = 0.02). Among subjects with T2DM, rs2268388 was not associated with BMI in Japanese (n = 2912) or EAs (n = 1149); however, the T allele associated with higher BMI in the subset with BMI≥30 kg/m(2) (n = 568 EAs; p-additive = 0.049, n = 196 Japanese; p-additive = 0.049). Association with BMI was strengthened in a T2DM meta-analysis that included an additional 756 AAs (p-additive = 0.080) and 48 Hong Kong Chinese (p-additive = 0.81) with BMI≥30 kg/m(2) (n = 1575; p-additive = 0.0033). The effect of rs2268388 on gene expression revealed that the T risk allele associated with higher ACACB messenger levels in adipose tissue (41 EAs and 20 AAs with BMI>30 kg/m(2); p-additive = 0.018) and ACACB protein levels in the liver tissue (mixed model p-additive = 0.03, in 25 EA bariatric surgery patients with BMI>30 kg/m(2) for 75 exams). The T allele also associated with higher hepatic triglyceride levels. These data support a role for ACACB in obesity and potential roles for altered lipid metabolism in susceptibility to DN.

Document Type

Article

Publication Date

2-27-2013

Notes/Citation Information

Published in PLoS ONE, v. 8, no. 2, e56193.

© 2013 Ma et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

Digital Object Identifier (DOI)

http://dx.doi.org/10.1371/journal.pone.0056193

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