Abstract

Oncogenic mutations activate secretory programs that drive tumor progression and represent therapeutic targets. The clinical relevance of CASPs is underscored by their prevalence, their linkages to specific genetic and epigenetic contexts, the proven efficacy of secretory blockade in preclinical models, and the availability of assays to identify vulnerable patient populations.

Document Type

Article

Publication Date

2024

Notes/Citation Information

© 2024, Tan et al. This is an open access article published under the terms of the Creative Commons Attribution 4.0 International License.

Digital Object Identifier (DOI)

https://doi.org/10.1172/JCI182652

Funding Information

This work was supported in part by grants from the NIH (R01CA236781 and R01CA255021-01 to JMK and 5R03CA280382-03 to XT). JMK holds the Gloria Lupton Tennison Distinguished Professorship in Lung Cancer.

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